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(11).
RECURRENT ACUTE PANCREATITIS AND PROGRESSION TO CHRONIC Recurrent acute pancreatitis and progression to chronic pancreatitis. 235.48 KB. 1. Introduction. Acute pancreatitis (AP) is a common clinical condition with a significant morbidity and mortality. AP has many causes and can be multifactorial. The role of genetic factors appears to be complex and is expanding as genetic mutations and their DRUG-INDUCED ACUTE PANCREATITIS TIMING OF ORAL REFEEDING AFTER ACUTE PANCREATITIS In a typical oral-refeeding protocol, the diet is reintroduced gradually, starting with small amounts of clear liquids for the first 24h. If tolerated, the diet is stepwise changed to a soft, low-fat regime followed by a solid diet. Hospital discharge is then contingent on tolerance of a low-fat solid diet (26). STEROID THERAPY IN THE MANAGEMENT OF AUTOIMMUNE In the initial inflammatory phase of AIP, the aim of treatment is to alleviate symptoms and improve radiological and biochemical abnormalities (13). In general, the indications for steroid therapy in type 1 AIP include symptoms, such as obstructive jaundice or abdominal pain, and the presence of symptomatic extrapancreatic lesions (6, 23).BÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm.LOGSDON, CRAIG D
Basic biology and physiology. Acute pancreatitis. Chronic pancreatitisSALUJA, ASHOK K.
Position/Title: Professor and Vice-Chair of Surgery, Director Pancreatic Cancer Research Institute.MARENINOVA, OLGA A.
11301 Wilshire Blvd, Bldg 258, Room 340. City: Los Angeles WELCOME TO THE PANCREAPEDIA Secretion of the Human Exocrine Pancreas in Health and Disease. Reviews. January 20, 2021. Darwin Conwell. Anatomy and Histology of the Pancreas. Reviews. January 18, 2021. Daniel S. Longnecker. Bioenergetics of the Exocrine Pancreas: Physiology to Pathophysiology. DIAGNOSIS AUTOIMMUNE PANCREATITIS Pancreatic histology obtained by core tissue biopsy (or a resected pancreatic specimen) is uniquely recognized as the “gold standard” for AIP diagnosis ( Histology of Autoimmune Pancreatitis ). This distinction is made on the basis that pathologists are able to accurately diagnose AIP independently of other clinical information(11).
RECURRENT ACUTE PANCREATITIS AND PROGRESSION TO CHRONIC Recurrent acute pancreatitis and progression to chronic pancreatitis. 235.48 KB. 1. Introduction. Acute pancreatitis (AP) is a common clinical condition with a significant morbidity and mortality. AP has many causes and can be multifactorial. The role of genetic factors appears to be complex and is expanding as genetic mutations and their DRUG-INDUCED ACUTE PANCREATITIS TIMING OF ORAL REFEEDING AFTER ACUTE PANCREATITIS In a typical oral-refeeding protocol, the diet is reintroduced gradually, starting with small amounts of clear liquids for the first 24h. If tolerated, the diet is stepwise changed to a soft, low-fat regime followed by a solid diet. Hospital discharge is then contingent on tolerance of a low-fat solid diet (26). STEROID THERAPY IN THE MANAGEMENT OF AUTOIMMUNE In the initial inflammatory phase of AIP, the aim of treatment is to alleviate symptoms and improve radiological and biochemical abnormalities (13). In general, the indications for steroid therapy in type 1 AIP include symptoms, such as obstructive jaundice or abdominal pain, and the presence of symptomatic extrapancreatic lesions (6, 23).BÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm.LOGSDON, CRAIG D
Basic biology and physiology. Acute pancreatitis. Chronic pancreatitisSALUJA, ASHOK K.
Position/Title: Professor and Vice-Chair of Surgery, Director Pancreatic Cancer Research Institute.MARENINOVA, OLGA A.
11301 Wilshire Blvd, Bldg 258, Room 340. City: Los Angeles DRUG-INDUCED ACUTE PANCREATITIS Frulloni, Luca. (2015). Drug-induced acute pancreatitis. 1. Introduction. Acute pancreatitis (AP) is a heterogeneous disease ranging from a clinically mild form to a more severe forms associated with high morbidity and mortality (78). A correct diagnosis of AP should be made within 48 h of admission. EPIDEMIOLOGY OF CHRONIC PANCREATITIS 1. Introduction. Epidemiologic descriptions of chronic pancreatitis (CP) have changed over time. The focus of reports from 1950-1990's was to describe the clinical profile and natural history in a series of patients. Many landmark studies during this period are crucial to our understanding of the disease. PAIN MANAGEMENT IN ACUTE PANCREATITIS Abstract. Severe abdominal pain is a hallmark of acute pancreatitis (AP). AP-associated pain is often described by patients as a deep and penetrating type of pain with acute onset and without any prodrome. Typically, AP patients locate the maximum of pain in the upper abdomen that radiates like a belt around the trunk into their back. CHOLECYSTOKININ TYPE 1 RECEPTOR Gene symbols: CCKAR 1. General Information. Cholecystokinin (CCK) exerts its physiological actions through the activation of two structurally-related class A G protein-coupled receptors (GPCRs) identified as type 1 CCK receptor (CCK1R) and type 2 CCK receptor (CCK2R) (also known as CCKAR and CCKBR, respectively, related to their prominent presence in “alimentary tract” and “brain”) (14 PANCREATOGENIC (TYPE 3C) DIABETES Pancreatogenic (Type 3c) Diabetes. 1. Definition. Pancreatogenic diabetes is a form of secondary diabetes, specifically that associated with disease of the exocrine pancreas. The most common disease of the exocrine pancreas associated with the development of diabetes is chronic pancreatitis. Analogous to chronic pancreatitis-associateddiabetes
CT AND MR FEATURES OF AUTOIMMUNE PANCREATITIS Introduction. Patients with autoimmune pancreatitis (AIP) often present with vague abdominal pain, jaundice, or weight loss. Differentiating AIP from pancreatic cancer is important to avoid unnecessary surgery or invasive intervention. When CT or MR reveals characteristic imaging findings of AIP, it is not difficult to make acorrect diagnosis.
GALPHA 12/13
Gene symbols: GNA12, GNA13 1. General Information. Gα 12/13 are the unique α subunits of a class of heterotrimeric G proteins along with Gα S, Gα i/o, and Gα q.α 12 and α 13 were initially cloned from a mouse brain cDNA library by PCR and show 67% amino acid identity with each other but only 35-40% with other Gα subunits (32). These α subunits are expressed in most tissues (34) andBÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm. MANAGEMENT OF ABDOMINAL COMPARTMENT SYNDROME IN ACUTE Abstract. The incidence of intra-abdominal hypertension (IAH) in patients with severe acute pancreatitis (SAP) is high (60-80% depending on the population studied). It is typically an early phenomenon, and caused by the inflammatory process in the pancreas aswell as
MARENINOVA, OLGA A.
11301 Wilshire Blvd, Bldg 258, Room 340. City: Los Angeles WELCOME TO THE PANCREAPEDIA Secretion of the Human Exocrine Pancreas in Health and Disease. Reviews. January 20, 2021. Darwin Conwell. Anatomy and Histology of the Pancreas. Reviews. January 18, 2021. Daniel S. Longnecker. Bioenergetics of the Exocrine Pancreas: Physiology to Pathophysiology. RECURRENT ACUTE PANCREATITIS AND PROGRESSION TO CHRONIC2 Recurrent acute pancreatitis and progression to chronic pancreatitis. 235.48 KB. 1. Introduction. Acute pancreatitis (AP) is a common clinical condition with a significant morbidity and mortality. AP has many causes and can be multifactorial. The role of genetic factors appears to be complex and is expanding as genetic mutations and their DIAGNOSIS AUTOIMMUNE PANCREATITIS Pancreatic histology obtained by core tissue biopsy (or a resected pancreatic specimen) is uniquely recognized as the “gold standard” for AIP diagnosis ( Histology of Autoimmune Pancreatitis ). This distinction is made on the basis that pathologists are able to accurately diagnose AIP independently of other clinical information(11).
TYPE 2 AUTOIMMUNE PANCREATITIS Type 2 AIP is characterized by the presence of GELs which are absent in type 1 AIP (1,19) . The second distinctive feature is the absence or only very low number (10 cells/hpf) IgG4 positive plasma cells intype 1
STEROID THERAPY IN THE MANAGEMENT OF AUTOIMMUNE In the initial inflammatory phase of AIP, the aim of treatment is to alleviate symptoms and improve radiological and biochemical abnormalities (13). In general, the indications for steroid therapy in type 1 AIP include symptoms, such as obstructive jaundice or abdominal pain, and the presence of symptomatic extrapancreatic lesions (6, 23). TIMING OF ORAL REFEEDING AFTER ACUTE PANCREATITIS In a typical oral-refeeding protocol, the diet is reintroduced gradually, starting with small amounts of clear liquids for the first 24h. If tolerated, the diet is stepwise changed to a soft, low-fat regime followed by a solid diet. Hospital discharge is then contingent on tolerance of a low-fat solid diet (26).LOGSDON, CRAIG D
Basic biology and physiology. Acute pancreatitis. Chronic pancreatitisBÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm.SALUJA, ASHOK K.
Position/Title: Professor and Vice-Chair of Surgery, Director Pancreatic Cancer Research Institute.MARENINOVA, OLGA A.
11301 Wilshire Blvd, Bldg 258, Room 340. City: Los Angeles WELCOME TO THE PANCREAPEDIA Secretion of the Human Exocrine Pancreas in Health and Disease. Reviews. January 20, 2021. Darwin Conwell. Anatomy and Histology of the Pancreas. Reviews. January 18, 2021. Daniel S. Longnecker. Bioenergetics of the Exocrine Pancreas: Physiology to Pathophysiology. RECURRENT ACUTE PANCREATITIS AND PROGRESSION TO CHRONIC2 Recurrent acute pancreatitis and progression to chronic pancreatitis. 235.48 KB. 1. Introduction. Acute pancreatitis (AP) is a common clinical condition with a significant morbidity and mortality. AP has many causes and can be multifactorial. The role of genetic factors appears to be complex and is expanding as genetic mutations and their DIAGNOSIS AUTOIMMUNE PANCREATITIS Pancreatic histology obtained by core tissue biopsy (or a resected pancreatic specimen) is uniquely recognized as the “gold standard” for AIP diagnosis ( Histology of Autoimmune Pancreatitis ). This distinction is made on the basis that pathologists are able to accurately diagnose AIP independently of other clinical information(11).
TYPE 2 AUTOIMMUNE PANCREATITIS Type 2 AIP is characterized by the presence of GELs which are absent in type 1 AIP (1,19) . The second distinctive feature is the absence or only very low number (10 cells/hpf) IgG4 positive plasma cells intype 1
STEROID THERAPY IN THE MANAGEMENT OF AUTOIMMUNE In the initial inflammatory phase of AIP, the aim of treatment is to alleviate symptoms and improve radiological and biochemical abnormalities (13). In general, the indications for steroid therapy in type 1 AIP include symptoms, such as obstructive jaundice or abdominal pain, and the presence of symptomatic extrapancreatic lesions (6, 23). TIMING OF ORAL REFEEDING AFTER ACUTE PANCREATITIS In a typical oral-refeeding protocol, the diet is reintroduced gradually, starting with small amounts of clear liquids for the first 24h. If tolerated, the diet is stepwise changed to a soft, low-fat regime followed by a solid diet. Hospital discharge is then contingent on tolerance of a low-fat solid diet (26).LOGSDON, CRAIG D
Basic biology and physiology. Acute pancreatitis. Chronic pancreatitisBÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm.SALUJA, ASHOK K.
Position/Title: Professor and Vice-Chair of Surgery, Director Pancreatic Cancer Research Institute.MARENINOVA, OLGA A.
11301 Wilshire Blvd, Bldg 258, Room 340. City: Los Angeles DRUG-INDUCED ACUTE PANCREATITIS Frulloni, Luca. (2015). Drug-induced acute pancreatitis. 1. Introduction. Acute pancreatitis (AP) is a heterogeneous disease ranging from a clinically mild form to a more severe forms associated with high morbidity and mortality (78). A correct diagnosis of AP should be made within 48 h of admission. PAIN MANAGEMENT IN ACUTE PANCREATITIS Abstract. Severe abdominal pain is a hallmark of acute pancreatitis (AP). AP-associated pain is often described by patients as a deep and penetrating type of pain with acute onset and without any prodrome. Typically, AP patients locate the maximum of pain in the upper abdomen that radiates like a belt around the trunk into their back. NATURAL COURSE OF CHRONIC PANCREATITIS 287.08 KB. 1. Introduction. In the minority of patients (i.e., 5.8% to 20%), chronic pancreatitis takes a primarily painless course (7, 8, 32, 52, 53, 76, 91). Exocrine and endocrine insufficiency are the dominating symptoms. For the majority of patients, however, pain is the decisive symptom, causing much discomfort in their daily lives. CHOLECYSTOKININ TYPE 1 RECEPTOR Gene symbols: CCKAR 1. General Information. Cholecystokinin (CCK) exerts its physiological actions through the activation of two structurally-related class A G protein-coupled receptors (GPCRs) identified as type 1 CCK receptor (CCK1R) and type 2 CCK receptor (CCK2R) (also known as CCKAR and CCKBR, respectively, related to their prominent presence in “alimentary tract” and “brain”) (14 MANAGEMENT OF ENDOCRINE FAILURE IN CHRONIC PANCREATITIS 1. Introduction. The natural history of chronic pancreatitis (CP) includes progressive loss of exocrine and endocrine function. Endocrine failure occurs due to progressive destruction of the gland by the ongoing inflammatory events of CP, and results in diabetes which is termed pancreatogenic or type 3c diabetes.GALPHA 12/13
Gene symbols: GNA12, GNA13 1. General Information. Gα 12/13 are the unique α subunits of a class of heterotrimeric G proteins along with Gα S, Gα i/o, and Gα q.α 12 and α 13 were initially cloned from a mouse brain cDNA library by PCR and show 67% amino acid identity with each other but only 35-40% with other Gα subunits (32). These α subunits are expressed in most tissues (34) andBÜCHLER, MARKUS W.
Professor Markus W. Büchler is the executive director and professor of surgery at the department of surgery at Heidelberg University. He studied medicine at the universities of Heidelberg and Berlin in Germany and underwent his surgical training in Berlin and at the university hospital in Ulm. MANAGEMENT OF ABDOMINAL COMPARTMENT SYNDROME IN ACUTE Abstract. The incidence of intra-abdominal hypertension (IAH) in patients with severe acute pancreatitis (SAP) is high (60-80% depending on the population studied). It is typically an early phenomenon, and caused by the inflammatory process in the pancreas aswell as
IMMUNOMODULATORS AND RITUXIMAB IN THE MANAGEMENT OF Steroid-sparing immunomodulators, such as azathioprine and mycophenolate mofetil, were primarily introduced in an effort to manage these patients. More recently, rituximab, a monoclonal anti-CD20 antibody, has also been used in these patients, including those who were resistant or intolerant to immunomodulators.MARENINOVA, OLGA A.
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